Poster Presentation Clinical Oncology Society of Australia Annual Scientific Meeting 2023

A 10 year experience of cardiac toxicity in patients receiving HER2 directed therapies at a tertiary Australian centre (#256)

Julia Freckelton 1 , Daphne Day 1 2 , Michelle White 1 , Piyumini Weerakoon Mudiyanselage 2 , Satish Ramkumar 1 , Sean Tan 1 2
  1. Monash Health, Clayton, VIC, Australia
  2. Medicine, Monash University, Clayton, Vic, Australia

Aims

HER2 directed therapies increase the risk of left ventricular (LV) dysfunction with reported incidence of asymptomatic LV dysfunction and congestive heart failure of ~10-20% and <4%, respectively. We performed a retrospective study in a major tertiary centre in Melbourne, Australia to characterise patterns and outcomes of cardiac toxicity resulting from HER2 directed therapy in patients with breast cancer.

Methods

Patients who completed 5 serial studies for LV assessment (baseline then 3-monthly) during the first 12 months of HER2 directed therapy from 2010-2020 were included. We recorded demographics, cancer history, cardiovascular risk factors and outcomes. LV dysfunction was defined as >10% reduction in LV ejection fraction (LVEF) to LVEF <50%.

Results

Of 518 patients who commenced HER2 directed therapy (trastuzumab +/- pertuzumab) during the study period, 222 (42.9%) completed recommended cardiac surveillance (all were female, median age 53 years,  145 (65%) prior anthracycline). Seventeen (7.7%) patients developed LV dysfunction (16 detected on surveillance; 13 asymptomatic, 4 symptomatic; 11 had \LVEF nadir45-50%, 6 of <45%). Of these, 9 had prior anthracycline chemotherapy and 10 had other cardiovascular risk factors. Thirteen patients had treatment interruption because of cardiac toxicity, 14 patients were referred to a cardiologist, 10 started angiotensin converting enzyme inhibitor treatment and 11, beta blocker therapy. Of these, 7 were successfully rechallenged.

 Conclusions

 Most patients treated with HER2 directed therapies did not undergo recommended cardiac surveillance in the first year of treatment. Among those that did, only a small proportion of patients developed symptomatic cardiac toxicity. Current surveillance guidelines are resource intensive and more prospective research is needed to determine the optimal cardiac surveillance frequency for these patients.